During a physical examination of the patient, the pulse rate is determined, since with aplasia, most often it is speeded up. First, the doctor takes a history and then conducts a physical examination of doxycycline. If bone marrow aplasia is suspected, the following examinations are prescribed: Blood test. Histological examination. cytogenetic study.



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However, a significant decrease in such cells is also observed in other diseases. Congenital bone marrow failure or myelodysplastic syndrome are common causes of hematopoietic hypoplasia. Therefore, further studies are needed to confirm the diagnosis.
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Cytogenetic testing may be required to rule out leukemia or myelodysplastic syndrome and other causes. Possible deviations in the number, as well as in the structure of chromosomes, can be detected using this research method. The acquired form of the disease is usually not characterized by defects in the genetic material. Detection of changes in chromosomes most likely indicates the presence of myelodysplastic syndrome.

If the factor causing anemia is known - radiation, chemicals, drugs - it is recommended to eliminate it. Treatment depends on the severity of the disease. Therapy for severe and very severe aplasia of the hematopoietic system is no different.

With a mild form of aplasia, expectant therapy is recommended: the patient should regularly visit a doctor and undergo routine examinations.

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The life expectancy of these patients does not decrease even in the absence of Doxycycline.

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In severe and very severe bone marrow aplasia, the following therapeutic measures are prescribed:

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Hematopoietic stem cell transplantation. immunosuppressive therapy. supportive therapy.

If there is a donor in the family (eg siblings), the bone marrow transplant should be performed as soon as possible. A long wait before transplantation and a large number of blood transfusions can worsen the patient's initial condition. If a suitable donor is not found, immunosuppressive therapy is prescribed. Preliminary planning of Doxycycline in a specialized center is absolutely necessary for bone marrow aplasia.

In an allogeneic transplant, the patient receives blood stem cells from another person. Blood cell precursors may belong to a relative or a stranger. Allogeneic transplantation from an unfamiliar donor is associated with relatively higher risks due to low tissue compatibility.
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  • Immunosuppressive treatment is given if a transplant cannot be performed.
  • If a suitable donor is not found, immunosuppressive therapy is recommended.
  • In this case, the combination of several drugs suppresses a defective immune response that has led to a decrease in blood-forming cells in the bone marrow.
  • In recent years, a combination of antithymocyte globulin and cyclosporine has been prescribed. During the first 4 days of inpatient treatment, antithymocyte globulin is administered through a vein.
  • In addition, patients receive a glucocorticoid agent for 4 weeks. As soon as the patient's health and blood counts improve, he can go home and be given medication in the form of a tablet or liquid.

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After immunosuppressive therapy, about 30% of patients experience a relapse of the disease. About 20% of patients develop acute myeloid leukemia or paroxysmal nocturnal hemoglobinuria. If in the first 3-6 months after the administration of the drugs there is no noticeable improvement in the composition of the blood, or after successful therapy, aplastic anemia recurs again, stem cell transplantation is necessary.

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Depending on the type of treatment (bone marrow transplantation or immunosuppressive drugs), differentSupportive measures that help fight the side effects or complications of the disease. Sometimes symptomatic remedies are prescribed to reduce fatigue.
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The chances of a patient recovering with timely treatment are quite high, although this is a life-threatening condition. Allogeneic transplantation of hematopoietic stem cells leads to the recovery of 80-90% of patients with hypoplasia in the bone marrow. A cell transplant from an unknown donor can also cure patients with bone marrow disease. However, many children and adolescents (about 20-30%) still develop serious and sometimes fatal complications.